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It targets inflammatory pathways and modulates/normalizes inflammatory mediators (e.g., TNF–α, INF-γ), systemic inflammatory biomarkers (e.g., RANTES), and other inflammatory cytokines [[158], [159], [163]]

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213 Introduction of HCK into each isoform allowed selective perturbation of activity to study interactions with other members of the p38/MAPK pathway, as well as to track crosstalk with proteins in other MAPK cascades such as the MEK/ERK pathway (Figure 10A)