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Importantly, both the collapse of ΔΨ m and arrhythmias are blocked by ligands of the mBzR complex, and not by cyclosporine A, the classical permeability transition pore (PTP) inhibitor, indicating a graded redox sensitivity of the target mitochondrial ion channels [6, 7]

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Wang et al, 1996) and in vivo (Wang et al, 1999a), or more recently in human colorectal cancer cells in vivo (Cusack et al, 2000)